Table 11.1 (continued)

Type of

NPs

Bacterial

strains/cells

Proposed mode of

action

Effect caused

Refs.

Au-

SPIONs

P. aeruginosa

NPs interact via

establishing an S-S

bonding with

cellular membrane

proteins

Disturbed cellular

metabolism

Inhibited redox

systems

Niemirowicz et al.

(2014)

SPIONs

–

ROS and

superoxide

production,

hydroxyl radical

formation,

oxidative stress,

catabolism of

carbon source and

generation of

nicotinamide

adenine

dinucleotide

(NAD)

Damaged cellular

macromolecules

(nucleic acids and

proteins)

Death of residual

bacteria

Lipid peroxidation

Bajpai and Gupta

(2011), Durmus

et al. (2012),

Hajipour et al.

(2012), and Leuba

et al. (2013)

Suppressed metabolic gene expression

TiO2

C3H10T1/

2 cells and

S. epidermidis

Physicochemical

properties

(elevated coronal

texture and

subordinate water

contact angle) and

chemical

constituents

(presence of

oxygen and

fluorine in

significantly

higher levels)

Diminished

adhesion and

colonization

(inhibited growth)

of pathogens on

NPs

Escalated adhesion

of C3H10T1/

2 cells on NPs

Decreased biofilm

formation

Peng et al. (2013)

and Roguska et al.

(2015)

OSM-2

Lactococcus

–

Increased

metabolic profiles

Altered bacterial

colonization

Enhanced

acetogenesis and

methanogenesis

due to an

enhancement in

the growth of

acetogenic bacteria

and archaebacteria

Reduced biofilm

formation

Pan et al. (2015)

11

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